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Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? for Software Development 2025 is part of Software Development preparation. The Question and answers have been prepared according to the Software Development exam syllabus. Information about Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? covers all topics & solutions for Software Development 2025 Exam. Find important definitions, questions, meanings, examples, exercises and tests below for Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer?.

Solutions for Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? in English & in Hindi are available as part of our courses for Software Development. Download more important topics, notes, lectures and mock test series for Software Development Exam by signing up for free.

Here you can find the meaning of Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? defined & explained in the simplest way possible. Besides giving the explanation of Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer?, a detailed solution for Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? has been provided alongside types of Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? theory, EduRev gives you an ample number of questions to practice Budding yeast cells that are deficient for Mad2, a component of the spindle-attachment-check point, are killed by treatment with benomyl, which causes microtubules to depolymerise. In the absence of benomyl, however, the cells are perfectly viable. Which explanation out of the following is able to justify this observation?a)In the absence of benomyl, the majority of spindles form normally and the spindle-attachment checkpoint (Mad2) plays no role.b)In the presence of benomyl, the majority of spindles form normally and Mad2 plays critical role in cell survival.c)Other than the role in cell survival, microtubule depolymerization affects oxidative phosphorylation in the absence of Mad2.d)Benomyl also affects protein synthesis in the absence of Mad2.Correct answer is option 'A'. Can you explain this answer? tests, examples and also practice Software Development tests.