Cysts of Entamoeba are formed in intestineof Man but these are dissolv...
Explanation:
Cysts of Entamoeba are formed in the intestine of Man but these are dissolved in the intestine of a new host (Man) because the inner layer of the cyst is not resistant to intestinal enzymes. Let's understand this in more detail:
Formation of Cysts of Entamoeba:
1. Entamoeba is a protozoan parasite that resides in the human intestine.
2. It exists in two forms: trophozoite and cyst.
3. The trophozoite form is the active form and is responsible for causing disease, while the cyst form is the dormant, infective form.
4. Under favorable conditions, the trophozoites multiply by binary fission in the intestine.
5. As the conditions become unfavorable, the trophozoites transform into cysts to protect themselves from harsh external conditions such as dehydration, low pH, and exposure to oxygen.
Structure of the Cyst:
1. The cyst of Entamoeba consists of two layers: the outer layer and the inner layer.
2. The outer layer is resistant to the harsh conditions outside the host, including exposure to digestive enzymes and other environmental factors.
3. The inner layer is susceptible to these conditions and can be dissolved by the digestive enzymes present in the intestinal tract.
Dissolution of Cysts in the Intestine of a New Host:
1. When the cyst is ingested by a new host, it passes through the stomach and reaches the intestine.
2. In the intestine, the cyst is exposed to the digestive enzymes secreted by the host's body.
3. The outer layer of the cyst protects it from the digestive enzymes, preventing its immediate dissolution.
4. However, the inner layer of the cyst is not resistant to these enzymes and gets dissolved, releasing the active trophozoites.
5. The trophozoites then attach to the intestinal wall and start multiplying, causing the infection.
Conclusion:
In summary, the cysts of Entamoeba are dissolved in the intestine of a new host (Man) because the inner layer of the cyst is not resistant to intestinal enzymes. This allows the release of active trophozoites, which can then cause infection and lead to the spread of the parasite.
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