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A Food and Drug lab has two new samples: a 240 gram cup of drip coffee, which contains 124 mg of caffeine, and a 60 gram cup of espresso, containing 160 mg of caffeine. If a technician were to create a new 120 gram cup sample that contained 50% coffee and 50% espresso, how many mg of caffeine would the new drink contain?
  • a)
    111
  • b)
    121
  • c)
    142
  • d)
    191
  • e)
    382
Correct answer is option 'D'. Can you explain this answer?
Most Upvoted Answer
A Food and Drug lab has two new samples: a 240 gram cup of drip coffee...
Understanding the Problem
To find the caffeine content in the new 120 gram cup sample that consists of 50% coffee and 50% espresso, we need to calculate the amount of caffeine contributed by each component.
Caffeine Content in Coffee
- The 240 gram cup of drip coffee contains 124 mg of caffeine.
- To find the caffeine concentration:
Caffeine per gram of coffee = 124 mg / 240 g ≈ 0.517 mg/g
- For a 60 gram portion of coffee (which is half of 120 g):
Caffeine from coffee = 60 g * 0.517 mg/g ≈ 31 mg
Caffeine Content in Espresso
- The 60 gram cup of espresso contains 160 mg of caffeine.
- Caffeine concentration in espresso:
Caffeine per gram of espresso = 160 mg / 60 g ≈ 2.67 mg/g
- For a 60 gram portion of espresso (which is also half of 120 g):
Caffeine from espresso = 60 g * 2.67 mg/g ≈ 160 mg
Total Caffeine in the New Sample
- Now, we sum the caffeine content from both coffee and espresso:
Total caffeine = Caffeine from coffee + Caffeine from espresso
Total caffeine = 31 mg + 160 mg = 191 mg
Conclusion
The new 120 gram cup sample, which contains 50% coffee and 50% espresso, will have a total caffeine content of 191 mg.
Thus, the correct answer is option D: 191 mg.
Free Test
Community Answer
A Food and Drug lab has two new samples: a 240 gram cup of drip coffee...
Understanding the Samples
To determine the caffeine content of the new 120-gram cup that is 50% coffee and 50% espresso, we first need to analyze the caffeine concentration in each original drink.
Caffeine in Drip Coffee
- Drip Coffee Weight: 240 grams
- Caffeine Content: 124 mg
Caffeine concentration in drip coffee:
Caffeine per gram = 124 mg / 240 g = 0.5167 mg/g
Caffeine in Espresso
- Espresso Weight: 60 grams
- Caffeine Content: 160 mg
Caffeine concentration in espresso:
Caffeine per gram = 160 mg / 60 g = 2.6667 mg/g
Calculating Caffeine in the New Sample
Since the new drink is a 120-gram mix of 50% coffee and 50% espresso, we have:
- Weight of Coffee: 120 g * 0.5 = 60 grams
- Weight of Espresso: 120 g * 0.5 = 60 grams
Caffeine from Coffee
Caffeine from 60 grams of coffee:
Caffeine = 60 g * 0.5167 mg/g = 31 mg
Caffeine from Espresso
Caffeine from 60 grams of espresso:
Caffeine = 60 g * 2.6667 mg/g = 160 mg
Total Caffeine in the New Drink
Total caffeine in the 120-gram drink:
Total Caffeine = Caffeine from Coffee + Caffeine from Espresso
Total Caffeine = 31 mg + 160 mg = 191 mg
Conclusion
Therefore, the new drink contains 191 mg of caffeine, which corresponds to option 'D'.
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Directions: Read the Passage carefully and answer the question as follow.Caffeine, the stimulant in coffee, has been called“the most widely used psychoactive substance on Earth .”Synder, Daly and Bruns have recently proposed thatcaffeine affects behavior by countering the activity in(5) the human brain of a naturally occurring chemical calledadenosine. Adenosine normally depresses neuron firingin many areas of the brain. It apparently does this byinhibiting the release of neurotransmitters, chemicalsthat carry nerve impulses from one neuron to the next.(10) Like many other agents that affect neuron firing,adenosine must first bind to specific receptors onneuronal membranes. There are at least two classesof these receptors, which have been designated A1 andA2. Snyder et al propose that caffeine, which is struc-(15) turally similar to adenosine, is able to bind to both typesof receptors, which prevents adenosine from attachingthere and allows the neurons to fire more readily thanthey otherwise would.For many years, caffeine’s effects have been attri-(20) buted to its inhibition of the production of phosphodi-esterase, an enzyme that breaks down the chemicalcalled cyclic AMP.A number of neurotransmitters exerttheir effects by first increasing cyclic AMP concentra-tions in target neurons. Therefore, prolonged periods at(25) the elevated concentrations, as might be brought aboutby a phosphodiesterase inhibitor, could lead to a greateramount of neuron firing and, consequently, to behav-ioral stimulation. But Snyder et al point out that thecaffeine concentrations needed to inhibit the production(30) of phosphodiesterase in the brain are much higher thanthose that produce stimulation. Moreover, other com-pounds that block phosphodiesterase’s activity are notstimulants.To buttress their case that caffeine acts instead by pre-(35) venting adenosine binding, Snyder et al compared thestimulatory effects of a series of caffeine derivatives withtheir ability to dislodge adenosine from its receptors inthe brains of mice. “In general,” they reported, “theability of the compounds to compete at the receptors(40) correlates with their ability to stimulate locomotion inthe mouse; i.e., the higher their capacity to bind at thereceptors, the higher their ability to stimulate locomo-tion.” Theophylline, a close structural relative of caffeineand the major stimulant in tea, was one of the most(45) effective compounds in both regards.There were some apparent exceptions to the generalcorrelation observed between adenosine-receptor bindingand stimulation. One of these was a compound called3-isobuty1-1-methylxanthine(IBMX), which bound very(50) well but actually depressed mouse locomotion. Snyderet al suggest that this is not a major stumbling block totheir hypothesis. The problem is that the compound hasmixed effects in the brain, a not unusual occurrence withpsychoactive drugs. Even caffeine, which is generally(55) known only for its stimulatory effects, displays thisproperty, depressing mouse locomotion at very low concentrations and stimulating it at higher ones.Q.According so Snyder et al, caffeine differs from adenosine in that caffeine

Directions: Read the Passage carefully and answer the question as follow.Caffeine, the stimulant in coffee, has been called“the most widely used psychoactive substance on Earth .”Synder, Daly and Bruns have recently proposed thatcaffeine affects behavior by countering the activity in(5) the human brain of a naturally occurring chemical calledadenosine. Adenosine normally depresses neuron firingin many areas of the brain. It apparently does this byinhibiting the release of neurotransmitters, chemicalsthat carry nerve impulses from one neuron to the next.(10) Like many other agents that affect neuron firing,adenosine must first bind to specific receptors onneuronal membranes. There are at least two classesof these receptors, which have been designated A1 andA2. Snyder et al propose that caffeine, which is struc-(15) turally similar to adenosine, is able to bind to both typesof receptors, which prevents adenosine from attachingthere and allows the neurons to fire more readily thanthey otherwise would.For many years, caffeine’s effects have been attri-(20) buted to its inhibition of the production of phosphodi-esterase, an enzyme that breaks down the chemicalcalled cyclic AMP.A number of neurotransmitters exerttheir effects by first increasing cyclic AMP concentra-tions in target neurons. Therefore, prolonged periods at(25) the elevated concentrations, as might be brought aboutby a phosphodiesterase inhibitor, could lead to a greateramount of neuron firing and, consequently, to behav-ioral stimulation. But Snyder et al point out that thecaffeine concentrations needed to inhibit the production(30) of phosphodiesterase in the brain are much higher thanthose that produce stimulation. Moreover, other com-pounds that block phosphodiesterase’s activity are notstimulants.To buttress their case that caffeine acts instead by pre-(35) venting adenosine binding, Snyder et al compared thestimulatory effects of a series of caffeine derivatives withtheir ability to dislodge adenosine from its receptors inthe brains of mice. “In general,” they reported, “theability of the compounds to compete at the receptors(40) correlates with their ability to stimulate locomotion inthe mouse; i.e., the higher their capacity to bind at thereceptors, the higher their ability to stimulate locomo-tion.” Theophylline, a close structural relative of caffeineand the major stimulant in tea, was one of the most(45) effective compounds in both regards.There were some apparent exceptions to the generalcorrelation observed between adenosine-receptor bindingand stimulation. One of these was a compound called3-isobuty1-1-methylxanthine(IBMX), which bound very(50) well but actually depressed mouse locomotion. Snyderet al suggest that this is not a major stumbling block totheir hypothesis. The problem is that the compound hasmixed effects in the brain, a not unusual occurrence withpsychoactive drugs. Even caffeine, which is generally(55) known only for its stimulatory effects, displays thisproperty, depressing mouse locomotion at very low concentrations and stimulating it at higher ones.Q.Snyder et al suggest that caffeine’s ability to bind to A1 and A2 receptors can be at least partially attributed to which of the following?(

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A Food and Drug lab has two new samples: a 240 gram cup of drip coffee, which contains 124 mg of caffeine, and a 60 gram cup of espresso, containing 160 mg of caffeine. If a technician were to create a new 120 gram cup sample that contained 50% coffee and 50% espresso, how many mg of caffeine would the new drink contain?a)111b)121c)142d)191e)382Correct answer is option 'D'. Can you explain this answer?
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