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Which component of blood is commonly referred to as "bad cholesterol" and is associated with the formation of artery-blocking plaques?
  • a)
    High-density lipoprotein (HDL)
  • b)
    Low-density lipoprotein (LDL)
  • c)
    Triglycerides
  • d)
    Hemoglobin
Correct answer is option 'B'. Can you explain this answer?
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Which component of blood is commonly referred to as "bad cholesterol" ...
Low-density lipoprotein (LDL) is often referred to as "bad cholesterol" and is associated with the formation of artery-blocking plaques.
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For some time, scientists have believed that cholesterol plays a major role in heart disease because people with familial hypercholesterolemia, a genetic defect, have six to eight times the normal level of cholesterol in their blood and they invariably develop heart disease. Scientists also noticed that people with familial hypercholesterolemia appear to produce more LDL’s (low-density lipoproteins) than normal individuals. However, scientists wondered, could a genetic mutation that causes a slowdown in the removal of LDL’s from the blood also result in an increase in the synthesis of this cholesterol-carrying protein? Since scientists could not experiment on human body tissue, their knowledge of familial hypercholesterolemia was severely limited. However, a breakthrough came in the laboratories of Yoshio Watanabe of Kobe University in Japan in 1980. Watanabe noticed that a male rabbit in his colony had ten times the normal concentration of cholesterol in its blood. By appropriate breeding, Watanabe obtained a strain of rabbits that had very high cholesterol levels. These rabbits spontaneously developed heart disease. To his surprise, Watanabe further found that the rabbits, like humans with familial hypercholesterolemia, lacked LDL receptors. What scientists learned by studying the Watanabe rabbits is that the removal of the VLDL (very low-density lipoproteins) remnant requires the LDL receptor. Normally, the majority of the VLDL remnants go to the liver where they bind to LDL receptors and are degraded. In the Watanabe rabbit, due to a lack of LDL receptors on liver cells, the VLDL remnants remain in the blood and are eventually converted to LDL’s. The LDL receptors thus have a dual effect in controlling LDL levels. They are necessary to prevent over synthesis of LDL’s from VLDL remnants and they are necessary for the normal removal of LDL’s from the blood.Q. According to the passage, by studying the Watanabe rabbits’ scientists learned that

For some time, scientists have believed that cholesterol plays a major role in heart disease because people with familial hypercholesterolemia, a genetic defect, have six to eight times the normal level of cholesterol in their blood and they invariably develop heart disease. Scientists also noticed that people with familial hypercholesterolemia appear to produce more LDL’s (low-density lipoproteins) than normal individuals. However, scientists wondered, could a genetic mutation that causes a slowdown in the removal of LDL’s from the blood also result in an increase in the synthesis of this cholesterol-carrying protein? Since scientists could not experiment on human body tissue, their knowledge of familial hypercholesterolemia was severely limited. However, a breakthrough came in the laboratories of Yoshio Watanabe of Kobe University in Japan in 1980. Watanabe noticed that a male rabbit in his colony had ten times the normal concentration of cholesterol in its blood. By appropriate breeding, Watanabe obtained a strain of rabbits that had very high cholesterol levels. These rabbits spontaneously developed heart disease. To his surprise, Watanabe further found that the rabbits, like humans with familial hypercholesterolemia, lacked LDL receptors. What scientists learned by studying the Watanabe rabbits is that the removal of the VLDL (very low-density lipoproteins) remnant requires the LDL receptor. Normally, the majority of the VLDL remnants go to the liver where they bind to LDL receptors and are degraded. In the Watanabe rabbit, due to a lack of LDL receptors on liver cells, the VLDL remnants remain in the blood and are eventually converted to LDL’s. The LDL receptors thus have a dual effect in controlling LDL levels. They are necessary to prevent over synthesis of LDL’s from VLDL remnants and they are necessary for the normal removal of LDL’s from the blood.Q. The passage supplies information to answer which of the following questions?

For some time, scientists have believed that cholesterol plays a major role in heart disease because people with familial hypercholesterolemia, a genetic defect, have six to eight times the normal level of cholesterol in their blood and they invariably develop heart disease. Scientists also noticed that people with familial hypercholesterolemia appear to produce more LDL’s (low-density lipoproteins) than normal individuals. However, scientists wondered, could a genetic mutation that causes a slowdown in the removal of LDL’s from the blood also result in an increase in the synthesis of this cholesterol-carrying protein? Since scientists could not experiment on human body tissue, their knowledge of familial hypercholesterolemia was severely limited. However, a breakthrough came in the laboratories of Yoshio Watanabe of Kobe University in Japan in 1980. Watanabe noticed that a male rabbit in his colony had ten times the normal concentration of cholesterol in its blood. By appropriate breeding, Watanabe obtained a strain of rabbits that had very high cholesterol levels. These rabbits spontaneously developed heart disease. To his surprise, Watanabe further found that the rabbits, like humans with familial hypercholesterolemia, lacked LDL receptors. What scientists learned by studying the Watanabe rabbits is that the removal of the VLDL (very low-density lipoproteins) remnant requires the LDL receptor. Normally, the majority of the VLDL remnants go to the liver where they bind to LDL receptors and are degraded. In the Watanabe rabbit, due to a lack of LDL receptors on liver cells, the VLDL remnants remain in the blood and are eventually converted to LDL’s. The LDL receptors thus have a dual effect in controlling LDL levels. They are necessary to prevent over synthesis of LDL’s from VLDL remnants and they are necessary for the normal removal of LDL’s from the blood.Q. The passage implies that Watanabe rabbits differ from normal rabbits in which of the following ways?

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Which component of blood is commonly referred to as "bad cholesterol" and is associated with the formation of artery-blocking plaques?a)High-density lipoprotein (HDL)b)Low-density lipoprotein (LDL)c)Triglyceridesd)HemoglobinCorrect answer is option 'B'. Can you explain this answer?
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